torsdag den 18. april 2013

Viktminskning är inte bara träning och kost....


Istället för att fokusera på att gå ner i vikt som sitt primära mål, pröva att ändra din inställning och attityd för att istället bli den bästa och sundaste version av dig själv. Att göra så resulterar i att man går ner i vikt och "fett-minskning" sker naturligt. Sann hälsa är mera än bara en sträng diet och träning. Överser du någon av dessa punkter??
  1. Hantera negativ stress och stress nivåerna i kroppen.
  2. Avlägsna mat man har intolerans för och som är inflammatorisk.
  3. Matsmältningsproblem, låga halter av HCL, enzymer och reparera mag- och tarm.
  4. Avslappning, avspänning och få sina 7-9 timmars sömn.
  5. Balansera hormoner, speciellt mellan insulin och cortisol.
  6. Intelligent träning. Styrketräning och intervallträning istället för konditionsträning, promenader.
  7. Att äta mat som har ett högt näringsvärde.
  8. Optimera lever funktionen.
  9. Reducera toxisk överbelastning från tungmetaller, kemikalier, xenoöstrogener etc
  10. Korrigera personlig näringsbrist
  11. Hantera underordnad och olösta emotionella problem och/eller trauma.
Att få framgångsrika viktminsknings resultat är en process. Det är en livsstil och inte en snabb-fix. Att väga sig hela tiden är det värsta man kan göra, då man ökar omedvetet stresshormonet kortisol i kroppen. 

Jag brukar att säga att man inte kan börja med att lägga nytt tak på det hus man köpt, om man fortfarande har mögel och svamp i källaren! Först måsta man ta bort möglet och torka källaren, lägga nytt golv och isolera väggarna, sätta i nya fönster och SÅ kan man lägga på ett nytt snyggt tak =) 

Vikt i Kg kan fördelas på olika vis, vilket vis väljer du? 

lørdag den 13. april 2013

Why is it important to take vitamin A-D-K and Zinc together?


Nutritional Adjuncts to the Fat-Soluble Vitamins A, D, and K


By Christopher Masterjohn
The “K” in “vitamin K” stands for “koagulation,” the German word for blood clotting. From its discovery in the 1930s through the late 1970s, we knew of no other roles for the vitamin.
The 1990s had come and nearly gone by the time awareness of its role in bone metabolism broke out of the confines of the vitamin K research community, and only in the twenty-first century has its role in preventing calcification of the blood vessels and other soft tissues become clear.
Vitamin K2, found in animal fats and fermented foods, is present in much smaller quantities in most diets when compared to vitamin K1, found in leafy greens.
Since researchers throughout the twentieth century saw the two forms of the vitamin as interchangeable, they ignored vitamin K2 as though its scarcity made it irrelevant.
The realization that vitamin K is not just for “koagulation,” however, led us to discover that vitamins K1 and K2 are not interchangeable after all: vitamin K1 more effectively supports blood clotting, while vitamin K2 more effectively ensures that calcium winds up in the bones and teeth where it supports health rather than in the soft tissues where it contributes to disease.
It was thus only in 2006 that the United States Department of Agriculture determined the vitamin K2 contents of common foods for the first time.1

Vitamins A, D, and K

While vitamin K2 languished in obscurity, vitamins A and D continually traded places with one another as the favored vitamin du jour. The pendulum initially swung in favor of vitamin D because rickets was common in the early twentieth century while eye diseases resulting from vitamin A deficiency were rare. It then swung in favor of vitamin A when that vitamin became known as the “anti-infective” vitamin.2
After World War II, the medical establishment had easy access to antibiotics and thus lost interest in battling infections with vitamin A.3 Vitamin D fared far worse, taking the blame for a British epidemic of infant hypercalcemia and eventually earning a reputation as “the most toxic of all the vitamins.”4 These days, the pendulum has swung full force in the opposite direction: we blame an epidemic of osteoporosis on vitamin A, and see vitamin D as the new panacea.5
Though a paradigm of synergy never took hold, it was not for want of opportunity. When Mellanby and Green first demonstrated in the 1920s that vitamin A prevented infections, they concluded that vitamin D could not be “safely substituted for cod-liver oil in medical treatment,” and that “if a substitute for cod-liver oil is given it ought to be at least as powerful as this oil in its content of both vitamins A and D.” 
Consistent with this point of view, clinical trials in the 1930s showed that cod liver oil could reduce the incidence of colds by a third and cut hours missed from work in half.6 Cod liver oil also caused dramatic reductions in mortality from less common but more severe infections. The medical establishment, for example, had been successfully using it to treat tuberculosis since the mid-nineteenth century.7
Studies in the 1930s expanded this to the treatment of measles.8 These findings made the popularity of cod liver oil soar (Figure 1).9
The idea that vitamin A alone was “antiinfective,” however, led to similar trials with halibut liver oil, which is rich in vitamin A but poor in vitamin D. These trials often failed to show any benefit. I.G. Spiesman of the University of Illinois College of Medicine proposed a simple solution to this paradox: vitamins A and D worked together to prevent infection, he suggested, and both vitamins are needed to prevent the common cold.
He published his own clinical trial in 1941, showing that massive doses of each vitamin alone provided no benefit and often proved toxic. Massive doses of both vitamins together, however, caused no toxicity and offered powerful protection against the common cold.10 Nevertheless, as antibiotics grew in popularity after World War II, interest in the fat-soluble vitamins waned and cod liver oil use began its steady decline (Figure 1).
The emergence of molecular biology in the late twentieth century provided new evidence for synergy. Vitamins A and D both make independent contributions to immune function by binding to their respective receptors and thereby directing cellular processes in favor of healthful immune responses, but studies in isolated cells suggest that vitamin D may only be able to activate its receptor with the direct cooperation of vitamin A.1112

We now know that vitamins A and D also cooperate together to regulate the production of certain vitamin K-dependent proteins. Once vitamin K activates these proteins, they help mineralize bones and teeth, support adequate growth, and protect arteries and other soft tissues from abnormal calcification, and protect against cell death.
As described below, the synergistic action of the fat-soluble trio depends on support from other nutrients like magnesium, zinc, fat and carbohydrate, as well as important metabolic factors such as carbon dioxide and thyroid hormone.
FIGURE 1:
Cod liver oil imports to the U.S., 1912-1965. Adapted from the data in reference 9.

Magnesium and the Fat-Soluble Vitamins

Magnesium contributes to more than three hundred specific chemical reactions that occur within our bodies, including every reaction that depends on ATP, the universal energy currency of our cells.13 Magnesium also activates the enzyme that makes copies of DNA, as well as the enzyme that makes RNA, which is responsible for translating the codes contained within our genes into the production of every protein within our body. This process of translating the DNA code in order to produce proteins is called “gene expression.”
Vitamins A and D carry out most of their functions by regulating gene expression, which means they rely directly on magnesium to carry out these functions. They also rely indirectly on magnesium because our cells can only produce their receptors and all the proteins with which they interact with the assistance of this critical mineral.
The well-studied interaction of magnesium with vitamin D and calcium provides an illustrative example. Magnesium is required for both steps in the activation of vitamin D to calcitriol, the form of vitamin D that regulates gene expression and stimulates calcium absorption. Even fully activated vitamin D (calcitriol), however, is useless in the absence of magnesium. Humans who are deficient in magnesium have low blood levels of both calcitriol and calcium, but treating them with calcitriol does nothing to restore calcium levels to normal. The only way to normalize calcium levels in these subjects is to provide them with sufficient magnesium. Magnesium also supports the cellular pumps that keep most calcium out of our soft tissue cells and make it available for the extracellular matrix of bones and teeth.
Altogether, these findings suggest that vitamins A and D can only fulfill their functions in the presence of adequate magnesium. As can be seen in Table 1, magnesium is abundant in many whole grains, nuts, seeds, legumes, and vegetables, some fruit, and some seafood. It is less abundant in meat, by contrast, and almost entirely absent from refined grains and sugar.14 Thus, we would expect a well-rounded diet devoid of “the displacing foods of modern commerce” to be rich in magnesium and thereby support the synergistic action of the fat-soluble vitamins.
winter2012masterjohntab1

Zinc and the Fat-Soluble Vitamins

As with magnesium, the fat-soluble trio can only support health if our diets contain adequate zinc. The interaction between vitamin A and zinc is particularly well studied.15 Vitamin A supports the intestinal absorption of zinc, possibly by increasing the production of a binding protein in the intestines. Zinc, in turn, supports the formation of vesicles involved in transporting vitamin A and the other the fat-soluble vitamins across the intestinal wall.
Zinc is an essential structural component of many vitamin A-related proteins, including the primary protein that transports vitamin A through the blood, the enzyme that carries out the first step in the activation of vitamin A to retinoic acid, and the nuclear receptor that binds to retinoic acid and allows it to regulate gene expression.
Numerous studies have demonstrated the interaction between zinc and vitamin A in humans. For example, in humans with marginal zinc status, zinc supplementation supports vitamin A’s role in visual function16 and eye development (Figure 2).17
Although less well studied, zinc also interacts with vitamin D. Vitamin D and zinc most likely promote each other’s intestinal absorption.18 In rats, dietary zinc supports the production of the vitamin D receptor.19 Once the receptor is formed, zinc provides it with essential structural support. Although in the absence of this structural support the receptor still binds to vitamin D, the structural support is needed to allow this vitamin-receptor complex to bind to DNA.20 Studies with isolated cells illustrate the importance of this interaction: adding zinc to these cells increases the rate at which vitamin D activates the expression of genes.21
Altogether, these results suggest that vitamins A and D can only fulfill their functions in the presence of adequate zinc. As can be seen in Table 2, zinc is found most abundantly in oysters, beef, and cheese.22 Chicken and eggs have considerably less zinc than beef, while milk and most plant foods contain very little. We absorb zinc about five times more effectively from animal products than from plant products. Thus, we should expect a diet that contains liberal quantities of zinc-rich animal products to support the synergistic action of the fat-soluble vitamins.
winter2012masterjohnfig2
Figure 2
Thirty-three Thai children ages six to thirteen with marginal zinc and vitamin A status were randomized in double-blind fashion to receive a placebo, 25 mg/day zinc, 1300 international units/day vitamin A, or a combination of the two supplements for six months. The figure shows the proportion of children with abnormalities in the conjunctiva of the eye, a characteristic sign of vitamin A deficiency. The synergistic interaction between zinc and vitamin A is statistically significant (P<0.02). The development of the surface of the eye and many other tissues is regulated by retinoic acid, the fully activated form of vitamin A. These results show that zinc supports the role of vitamin A in tissue development, probably by supporting the activity of enzymes involved in activating vitamin A to retinoic acid and by allowing the nuclear receptors for retinoic acid to bind to DNA and regulate gene expression. Adapted from reference 17.
winter2012masterjohntab2

Fat, Carbs, Thyroid and Carbon Dioxide

In order to absorb fat-soluble vitamins from our food, we need to eat fat. Human studies show that both the amount and type of fat are important. For example, one study showed that absorption of beta-carotene from a salad with no added fat was close to zero. The addition of a lowfat dressing made from canola oil increased absorption, but a high-fat dressing was much more effective.23 Canola oil, however, is far from ideal. Studies in rats show that absorption of carotenoids is much higher with olive oil than with corn oil.24
Similarly, studies in humans show that consuming beta-carotene with beef tallow rather than sunflower oil increases the amount we absorb from 11 to 17 percent. The reason for this is unknown, but it may be that oils rich in polyunsaturated fatty acids promote the oxidative destruction of fat-soluble vitamins in the intestines before we are able to absorb them. Thus, the more fat we eat, and the lower those fats are in polyunsaturated fatty acids, the more fat-soluble vitamins we absorb.
While dietary fat is clearly important, there may be a role for dietary carbohydrate as well. Once vitamins A and D stimulate the production of vitamin K-dependent proteins, vitamin K activates those proteins by adding carbon dioxide to them. Once added to a protein, carbon dioxide carries a negative charge and allows the protein to interact with calcium, which carries a positive charge. The greater the supply of carbon dioxide, the better vitamin K can do its job.25 Carbohydrates are rich in carbon and oxygen, and when we break them down for energy we release these elements in our breath as carbon dioxide. Because carbohydrates are richer in oxygen, burning them generates about 30 percent more carbon dioxide per calorie than burning fat, and low-carbohydrate diets have been shown to lower blood levels of carbon dioxide (Figure 3).26
Ideally, we should study this further by determining whether dietary carbohydrate affects the amount of activated vitamin K-dependent proteins in humans.
We also produce more carbon dioxide when we burn more calories, regardless of whether we are burning carbohydrate or fat. Intense exercise more than doubles the amount of carbon dioxide we produce compared to what we produce when at rest.27Even working at a standing desk rather than a sitting desk increases both calories burned and carbon dioxide generated by about a third (Figure 4).28
Future studies should directly investigate whether exercise increases the activation of vitamin K-dependent proteins, but it seems reasonable to suggest that part of the reason exercise promotes cardiovascular health may be because it ensures a more abundant supply of carbon dioxide, which vitamin K uses to activate proteins that protect our heart valves and blood vessels from calcification. Thyroid hormone is a key regulator of the metabolic rate and may thus be a major determinant of the carbon dioxide available for activating vitamin K-dependent proteins. Theoretically, thyroid hormone should increase the rate of metabolism and a greater rate of metabolism should produce a proportionally greater supply of carbon dioxide.
Thyroid hormone directly increases the production of vitamin K-dependent proteins and protects blood vessels from calcification in rats.29 The reason for this relationship is unclear. We could speculate, however, that our bodies in their infinite wisdom use thyroid hormone to tie the production of vitamin K-dependent proteins to the production the carbon dioxide needed to activate them.
winter2012masterjohnfig3
Figure 3: Low-Carbohydrate Diets Reduce Blood Levels of Carbon Dioxide
Investigators fed twenty adult patients requiring artificial ventilation a standard or low-carb, high-fat diet through a feeding pump. The purpose of the study was to determine whether a low-carbohydrate diet could be used to reduce the need for a ventilator. Carbohydrates generate more carbon dioxide than fat, and we eliminate excess carbon dioxide by breathing faster. The study showed that patients on a low-carbohydrate diet generated less carbon dioxide, breathed more slowly, and required less time on a ventilator. Nevertheless, for healthy individuals who do not require artificial ventilation, dietary carbohydrates may support the activity of vitamin K, which activates certain proteins by adding carbon dioxide to them. Adapted from reference 26.
winter2012masterjohnfig4
Figure 4:
Twenty healthy young adult men and women performed crossword puzzles and word finds for forty-five minutes either at a traditional sitting desk or at a standing desk. Adapted from reference 28

The Big Picture

It is clearly time to move beyond viewing each vitamin in isolation. The fat-soluble vitamins not only synergize with each other, but cooperate with many other nutrients and metabolic factors such as magnesium, zinc, fat, carbohydrate, carbon dioxide and thyroid hormone.
This paradigm has two important implications. At the level of scientific research, a study about one vitamin can easily come to false conclusions unless it takes into account its interactions with all the others. We should reverently and humbly bow before the complexity of these interactions, realizing how little we know and recognizing that we are always learning. At the level of personal health, these interactions emphasize the need to consume a well-rounded, nutrient-dense diet. Supplementation with an individual vitamin runs the risk of throwing it out of balance with its synergistic partners. The fat-soluble vitamins work most safely and effectively when we obtain them from natural foods within the context of a diet rich in all their synergistic partners.

Zinc and the Dark Adaptation Test for Vitamin A Deficiency

The role of vitamin A in vision is unusual. This vitamin carries out most of its known actions by regulating the expression of specific sets of genes. Vitamin A regulates gene expression only after being activated in a two-step process from retinol to retinal, and finally to retinoic acid. Vitamin A supports vision, however, in its semi-activated form as retinal. Retinal binds to a protein known as opsin, forming a vitamin-protein complex known as rhodopsin. Each photon of light that enters our eye and collides with rhodopsin causes the retinal to change shape and release itself from the complex. This event then translates into an electrical impulse that our optic nerve transmits to our brain. The brain synthesizes myriad such electrical impulses at every moment and interprets them as vision.30
While the function of opsin is to help generate visual images by binding and releasing vitamin A, opsin can only maintain its proper shape and function when it is bound to zinc. In addition, zinc supports the conversion of retinol to retinal, the form of vitamin A that binds to opsin. We could predict, then, that vitamin A would only be able to support vision in the presence of adequate zinc. This can be studied by determining dark adaptation thresholds, which determine the dimmest spots of light we are able to see after having spent a period of time in the dark to maximize our visual sensitivity. When vitamin A is insufficient, we lose the ability to see the dimmer spots of light.
Robert Russell of Tufts University studied ten patients with deficient blood levels of vitamin A who also failed the dark-adaptation test. Eight of them achieved normal dark-adaptation thresholds after supplementing with 10,000 international units of vitamin A for two to four weeks. Two of them, however, had deficient blood levels of zinc. Vitamin A supplementation alone failed to normalize their visual function, but adding 220 milligrams per day of zinc to the regimen for two weeks brought it back to normal.16 These results show that vitamin A can only support healthy vision with the direct assistance of zinc.

About the Author

Chris Masterjohn has a PhD in nutritional sciences from the University of Connecticut, and has published five peer-reviewed papers on vitamins and supplementation. He researches fat-soluble supplements – A, D, and K – at the University of Illinois. He also maintains a blog, The Daily Lipid, and his website, Cholesterol-And-Health.com, which are dedicated to the issue of cholesterol. He's also active with the Weston A. Price Foundation.

fredag den 5. april 2013

Forvirring om rød kødt og inflammation.

Som ernæringsfysiolog tænker jeg på en helt anden måde en Kris skriver om i denne artikel. Han skriver at vi som mennesker har spist kød i tusind vis af år og at Masai folkets diæt består næsten kun af kød, mælk og blod, det er også rigtigt og at de ikke havde nogle symptomer på inflammation eller cancer. Det er da helt klart!!!
1) Det kød de spiste for tusind vis af år siden og som Masia folker spier er 100% økologisk, køerne spiser kun gras og mælken fra køerne er rå, ikke homegineseret eller pasurisert. Blodet er ikke forurenet med anti-bitoka, steroider eller andre vækst faktorer fra  kraft foder. De varmer deres kød over ild uden dårlige fedstoffer, og de spiser det ikke sammen med frensh fries!
2) Vores forfædre og Masia folket gik eller løb flere kilometer hver dag for at kunne finde mad!! De havde en livsstil som gjorde at de kunne tåle at spise disse rene produkter og de fik masser af frisk luft når de gik over savannen. 
3) Kød fra konventionelle køer der får kraftfoder (GM-soja, etc), anti-biotika og anden medicin har også et lavere indhold af omega 3 en de køer der går frit og spiser gras. Det indholder også østrogener. Alt dette optages in i vores blod, celler og DNA når vi spiser kød fra disse køer. Når man så steger eller griller kød så det bliver sort dannes kraftig cancer fremkaldende stoffer og hvis vi så steger i billig opvarmet olie som gennem opvarmningen har ændret form til transfetsyrer så øger vi også vores chancer at få åreforkalkning pga at det dårlige LDL kolesterol oxiderer (ilter) i varmen og sætter sig fast i karvæggene og dermed danner "åreforkalkning"-åreforfettning og risikoen for blodpropper øger.
4) Hvis man så også spiser "vores" kød som hamburger med en hveddbolle (gluten), ketchup (sukker) og mayonæse (transfet), drikker nogle øl eller sodavand, ryger (alt dette øger chancen at danne inflammation) og samtidigt sidder i sofaen og kigger i fjernsyn, så kan I nok også se forskellen på kød og kød og inflammation eller ikke!!!
5) Vores forfæder havde ikke luftforurening, cigaretter, alkohol, tv og en sofa at fede den af på, heller ikke sodavand og alle sociale problemer vi har idag eller stress som vi kender den fra arbejde og famile liv.
Så man kan ikke sige at bare fordi at vi som mennesker har spist kød i tusindvis af år og drukket mælk så kan vi også gøre det idag. Det er to helt forskellige ting: 
1) Økologisk kød fra frit gående køer som kun spiser gras, nul luftforurening, masser af motion, ingen stress (som vi kender den idag) frisk vand og frisk luft, ikke nogle mediciner = En naturlig del af livet, giver ikke sygdom.
2) Behandlat kød fra konvetionelle køer med hormon- og antibiotika behandling, pasteuriseret og homogeniseret mælk (som kroppen ikke kan genkende, det er ikke mad), luft- og cigaretforurening (gifter), alkohol, medicin, stille siddende livsstil-dovenhed og stress = Giver meget og forskellige sygdomme pga inflammation. Vores livsstilssygdomme vi har i dag, kræft, diabetes, fedme, højt blodtryk, åreforkalkning og blodprop etc.
Hvis man vælger at spise kød og drikke mælk, så sørg for at det er økologisk kød og ikke homogeniseret- og eller lav pasteuriseret mælk fra økologisk dyrkning. Sørg for at ikke ryge, overdriven alkohol indtagelse og motionere 3-4 ggr i ugen, meditere og bliv fri fra stress og nyd livet. =) 
Her er artikeln: 
"Two different controlled trials have measured inflammation markers in response to increased red meat intake, and both have found that red meat does not elevate these markers. The first study concludes that increasing red meat consumption by replacing carbohydrates in the diet of non-anemic individuals actually reduces markers of inflammation. (1) The other study showed that in anemic women, inflammation markers on a diet high in red meat were not significantly different from those on a diet high in oily fish. (2) This evidence suggests that red meat is not more inflammatory than other meats for most people, and is potentially less inflammatory than dietary carbohydrates. However, I’d like to discuss a couple of other specific mechanisms that are often blamed for inflammation."

Neu5Gc

Despite the lack of controlled trials demonstrating that red meat is inflammatory, there has been recent concern over a compound in red meat called Neu5Gc. (3) Neu5Gc is a monosaccharide that acts as a type of signaling molecule in mammalian cells, and one of its functions is to help the immune system distinguish between ‘self’ cells and ‘foreign’ cells. (4) Humans lost the ability to produce Neu5Gc millions of years ago through a genetic mutation, although we still produce the closely related compound Neu5Ac. (5) Humans are unique in this respect, because most other mammals still produce Neu5Gc, which is why that compound is found in mammalian meat.
When humans consume red meat and milk products, we incorporate some of this compound into our own tissues, especially tissues that grow at a fast pace such as fetuses, epithelial and endothelial tissue, and tumors. (6) The concern is that most of us also have anti-Neu5Gc antibodies circulating in our blood, and some researchers have suggested that these antibodies react with the Neu5Gc in our tissues to create chronic inflammation, leading to chronic diseases such as cancer.
The problem is that researchers are nowhere near proving that hypothesis. Research is in the very earliest stages, and while some fascinating hypotheses involving this molecule are being generated, the studies needed to confirm or refute these hypotheses are nonexistent. Most of the studies done on the topic acknowledge that at this point, any role in chronic inflammation is speculative, but many who have cited their research neglect to acknowledge that limitation. Thus begins a new round of fear mongering at the expense of red meat.
In the absence of conclusive evidence one way or another, it can be helpful to remember that red meat has been part of the human diet for much of our history, and remains an important dietary element of many healthy cultures. For example, the traditional diet of the Masai was composed almost entirely of red meat, blood, and milk – all high in Neu5Gc – yet they were free from modern inflammatory diseases. (7) If Neu5Gc really caused significant inflammation, the Masai should’ve been the first to know, because they probably couldn’t have designed a diet higher in Neu5Gc if they tried.

Arachidonic Acid

Arachidonic acid (AA) is often cited as a source of inflammation, and because AA is found primarily in eggs and meat, this concern could contribute to the view that red meat is inflammatory. AA is an essential omega-6 fatty acidthat is a vital component of cell membranes and plays an important role in the inflammatory response. (8) It’s especially necessary during periods of bodily growth or repair, and is thus a natural and important component of breast milk. (9) AA is sometimes portrayed as something to be avoided entirely simply because it is ‘inflammatory,’ but as usual, that view drastically oversimplifies what actually happens in the body.
It’s true that AA plays a role in inflammation, but that’s a good thing! It ensures that our body responds properly to a physical insult or pathogen, and it also helps ensure that the inflammatory response is turned off when it’s no longer needed. AA interacts with other omega-3 and omega-6 fatty acids in intricate and subtle ways, and an imbalance in any of those fats has undesirable effects. For example, low doses of EPA tend to increase tissue levels of AA, while high doses decrease levels of AA, which probably explains why the benefits of fish oil supplementation are lost at higher doses. (10)  In epidemiological studies, higher plasma levels of both AA and the long-chain omega-3 PUFA were associated with the lowest levels of inflammatory markers. (1112) And clinical studies have found that adding up to 1,200 mg of AA per day—which is 12 times higher than the average intake of AA in the U.S.— to the diet has no discernible effect on the production of inflammatory cytokines. (1314) What’s more, our Paleolithic ancestors (who were largely free of chronic, inflammatory disease) consumed at least twice the amount of AA that the average American does today. (15)
Finally, it’s important to note that red meat actually has a lower concentration of AA than other meats because of its lower overall PUFA content. (16)(17) Additionally, red meat has been shown to increase tissue concentrations of both AA and the long chain omega-3s DHA and EPA, preserving the all-important balance of omega-3 and omega-6. (18)

Charred meat and cancer

The final concern I want to address involves compounds that are produced when meat is cooked, including advanced glycation end products (AGEs), heterocyclic amines (HAs), and polycyclic aromatic hydrocarbons (PAHs). Again, this applies to all meat, not just red meat, but it can still contribute to the perception that red meat is unhealthy.
HAs and PAHs have both been shown to cause cancer in animal models, and although these results can’t necessarily be extrapolated to humans, it’s probably wise to limit exposure to these two compounds. (19)(20) HAs and PAHs are formed when meat is cooked using high-heat or dry cooking methods such as frying, grilling, or smoking. But while cooked meat is the only significant source of HAs, PAHs are a ubiquitous environmental contaminant, and the bulk of dietary PAHs actually come from vegetables and grains. (21) In fact, levels of PAH in leafy vegetables are comparable to levels in smoked meat! However, the highest food levels of PAH are found in charred meats that have been cooked over an open flame.
AGEs are different from the other compounds in that they can be formed both endogenously and exogenously. (22) Like HAs and PAHs, AGEs are formed when foods – particularly meat – are cooked, although they are also naturally present even in uncooked meat. However, dietary AGEs do not tell the whole story, because they can also form through various metabolic pathways in the body. One study showed that while omnivores generally have higher dietary intakes of AGEs than vegetarians, vegetarians actually end up with higher concentrations of AGEs in their plasma. (23) The authors hypothesized that their results were due to the increased fructose intake of vegetarians, although another plausible mechanism appears to be the inhibition of AGE formation by carnosine, an amino acid found in meat. (24)(25) Either way, I wouldn’t be terribly concerned about AGEs in meat, although I still recommend favoring lower-heat cooking methods to avoid HAs and PAHs.
If you do want to grill or fry your meats, you can significantly reduce the formation of all of these compounds by using an acidic marinade, which has the added bonus of tasting great! Marinating beef for one hour reduced AGE formation by over half, and marinades can cut HA formation in meat by up to 90%. (26)
Overall, there’s no good evidence that red meat is more inflammatory than other meats, and some evidence indicating that it’s less inflammatory. Just like any other food, it’s certainly possible for people to have individual intolerances to red meat that might induce inflammation, but there’s no reason for most people to restrict red meat on the basis of inflammation. Additionally, AGEs from meat are probably not a concern, and meat eaters might even be better off when it comes to plasma levels of AGEs. Any concerns about other compounds produced by cooking meat can be minimized simply by favoring wet or low-heat cooking methods, or using a marinade when high-heat methods are desired. Se hele artikel her: 

Kilde: 

http://chriskresser.com/does-red-meat-cause-inflammation?fb_action_ids=10151591453352489&fb_action_types=og.likes&fb_source=other_multiline&action_object_map=%7B%2210151591453352489%22%3A505362676192424%7D&action_type_map=%7B%2210151591453352489%22%3A%22og.likes%22%7D&action_ref_map=%5B%5D

lørdag den 19. januar 2013

Holistic approach to stress and panic attacks

I found this fantastic article on Authentic Discovery about how to relieve stress, panic attacks and anxiety with EFT techniques. Good reading and remember, we are all energy, every single cell has molecules that are single atoms that are full of life with a bundle of energy inside of then (protons, electrons, photons and neutrons). / Sofie =)


I AM statementsPDFPrintE-mail
Written by Kathy Hughes   
Friday, 30 March 2012 10:56

 
 

When we don’t feel good about something which has happened to us, we tend to create the event over and over in our minds. Thus it becomes harder to escape from the uncomfortable feelings associated with our story.  We then become addicted to the drama by giving it more energy, and the true self gets hidden amongst it.
Look at the 'I AM' table and notice which statement stands out the most. If it is a negative belief, are you feeling this now? 
Your true essence is light but when we focus on our problems and allow them to grow our light becomes dulled. Thus the negative energy we have created then becomes more dominant. The statements on the right of the 'I AM' chart are your true authentic self.
i am  
Imagine your inner light is shining where there is only solutions, love, honour and knowledge.  Honour any persons involved and honour the situation. You will be given the resources and strength to deal with the problem from interesting circumstances that will fill you with wonder.
Sometimes awareness can help to defuse the situation and uncomfortable feelings. Other times we may need some extra help by discussing it with a friend or with a good practitioner. By writing down your concern or worry, can often bring clarity and peace. An awesome site,www.itsaworry.com is a perfect place to enter your worry and give clarity. Try it and observe an interesting twist as you see how your story unfolds in a more positive light.  www.itsaworry.com

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